Pedro San Cristobal principal interest is in human arterial hypertension, and its regulation by ion transport processes in kidney, particularly DCT, in health and disease. He joined the unit in March 2009. Actually his current projects involve the molecular mechanisms controlling the sodium, calcium and magnesium homeostasis. Functional analysis of mutations in the human, NKCC1/2, NCC and Kv1.1 identified as causatives of Bartter and Gitelman syndrome and inherited hypomagnesemia respectively. His studies include use of diverse models established epithelial cell lines, X. laevis ocytes, tissue-specific mice and biochemical analysis of protein activity.